Hepatitis means inflammation of the liver. Toxins, certain drugs, some diseases, heavy alcohol use, and bacterial and viral infections can all cause hepatitis. Hepatitis is also the name of a family of viral infections that affect the liver; the most common types in the United States are hepatitis A, hepatitis B, and hepatitis C.
Hepatitis B is a contagious liver disease that results from infection with the hepatitis B virus. It can range in severity from a mild illness lasting a few weeks to a serious, lifelong illness. Hepatitis B is usually spread when blood, semen, or another body fluid from a person infected with the hepatitis B virus enters the body of someone who is not infected. This can happen through sexual contact with an infected person or sharing needles, syringes, or other drug-injection equipment. Hepatitis B can also be passed from an infected mother to her baby at birth.
Hepatitis B can be either acute or chronic. Acute hepatitis B virus infection is a short-term illness that occurs within the first 6 months after someone is exposed to the hepatitis B virus. Acute infection can — but does not always — lead to chronic infection. Chronic hepatitis B virus infection is a long-term illness that occurs when the hepatitis B virus remains in a person’s body. Chronic hepatitis B is a serious disease that can result in long-term health problems, and even death.
Hepatitis B Universal Vaccination: Learning from the French Experience
The First Cancer Vaccine: Facts and Failings by F. Edward Yazbak, MD, FAAP
Hepatitis B: Discrimination and Vaccine Damage:
For more than 20 years, the National Vaccine Information Center (NVIC) has been receiving reports that adults, children and infants are suffering serious reactions to hepatitis B vaccine. Hepatitis B vaccine reaction reports began to come in to NVIC’s Vaccine Reaction Registry in the late 1980’s from adult health care workers, who reported extreme fatigue, muscle weakness, joint pain, loss of vision, memory loss, heart problems, and development of multiple sclerosis after hepatitis B vaccination.
Infants who contract hepatitis B, either from their infected mother or infected blood transfusions, are at highest risk for chronic infection. But for the majority of healthy teenagers and adults, who come down with hepatitis B infection, symptoms include nausea, vomiting, low grade fever, pain and swelling in the joints, headache, and cough for two weeks before the onset of jaundice and enlargement/tenderness of the liver that lasts for three to four weeks. Fatigue can last up to a year but “most patients do not require hospital care” and “95 percent of patients have a favorable course and recover completely” with the case fatality ratio being “very low (approximately 0.1 percent)” according to Harrison’s Principles of Medicine (11th Edition).
Those who recover completely from hepatitis B infection acquire life-long immunity. One medical textbook (Robbins Pathologic Textbook of Disease) points out that among those who do not recover completely from hepatitis B infection, fewer than 5 percent become chronic carriers of the virus, with just one quarter of these in danger of developing life threatening disease later in life.
There is no reason a pregnant mother can’t be tested for Hepatitis B before she delivers, or after delivery, and have her baby tested as well in lieu of a vaccine given at birth. Some states mandate testing pregnant mothers before birth as well, so you would know ahead of time if you were positive or negative. If a mother is positive, the baby would be given hepatitis B immue globulin (HBIG).
Hepatitis B and Titers
One feature of the Hepatitis B is that if you never show antibodies, there is no point in having more shots. Some people may even test false positive with titers.
There are two groups of people:
1. Those who have an immune response to hepatitis B that is weak, and have difficulty clearing the virus from their system. These are the people who may become carriers, and whose responses to the vaccine are naturally stunted as well. Booster shots in this group would be short lived.2. For others, the immune system gets rid of the virus without a problem. They produce higher titers, and even if their detectable antibodies become next to nothing, they will still respond well to re-infection or a vaccine booster. They are also the group of people who would never become carriers or get cancer in the first place.Basically, the people who need the vaccine the most because they would be most likely carriers or get cancer, is the group least likely to respond well to the vaccine, and to have the poorest response to boosters.In a nut shell- those who don’t need the vaccine, respond best. Those who do need it, respond the worst.
If 30-50% of persons who develop adequate antibody after 3 doses of the vaccine, loose detectable antibody within 7 years, by the time a child is 7 years old, they won’t show any benefits of the vaccine! (Parent Information. Understanding infant hepatitis B immunisation. Immunise Australia program, NHMRC) Further more, carriers can be carriers for over 40 years and not know until they have a blood test that tells them. People, who are carriers that live clean lives, don’t drink alcohol, and maintain a good diet, carriage will mean nothing as it will not be a problem in their lives. It will only be a problem if there was a second, third and beyond tiers done of co-carcinogens to trigger the virus into creating potential cirrhosis and subsequent cancer. If the parents have no antibodies to the disease, how do they know the vaccine will give their children antibodies? When you read medical literature on carriers, you will see it runs in families because there is an immune defect that prevents the body from either clearing it, or creating antibodies.
A study in Science Direct:
Short-term response to a booster dose of hepatitis B vaccine in anti-HBs negative adolescents who had received primary vaccination 16 years ago:
We conducted a revaccination study to investigate the short-term response to booster hepatitis B (HB) vaccination in seronegative adolescents who had received primary infantile HB vaccination. A booster dose of recombinant HB vaccine was administered to 395 adolescents 15–18 years of age whose serum titers of antibody against hepatitis B surface antigen (HBsAg) (anti-HBs) were <10 mIU/mL. Seventy-seven percent of the booster recipients converted to anti-HBs seropositivity (postbooster titers ≥ 10 mIU/mL). As compared with adolescents who had undetectable prebooster anti-HBs titers (<0.1 mIU/mL), the seropositive rates and geometric mean titers (GMTs) of 2-month and 1-year postbooster were significantly higher for those of prebooster titers of 0.1–0.9 and 1.0–9.9 mIU/mL (all p < 0.0001). Postbooster titers declined significantly more rapidly for those with undetectable prebooster anti-HBs titers than for those with prebooster titers of 0.1–0.9 and 1.0–9.9 mIU/mL. Our observations indicate that a booster dose of HB vaccine maybe unable to induce sufficient immunological response in adolescents who had undetectable residual anti-HBs titers.
So, that means all the babies being vaccinated now will have to do the whole series again when they are older and might have a small risk of developing Hepatitis B. Tell me again what was the point in vaccinating all babies from birth again???
Filed under: Hepatitis B |