Antibodies Do NOT Produce Immunity


Antibodies do not produce immunity. 

The immune system consists of at least two parts which are the humoral and the cellular. When one is activated the other is suppressed. Because of this, the new approach has been to try and prevent suppression.


Dr. Rebecca Carly explains:

The mechanism by which the immune system is corrupted can best be realized when you understand that the two poles of the immune system (the cellular and humoral mechanisms) have a reciprocal relationship in that when the activity of one pole is increased, the other must decrease. Thus, when one is stimulated, the other is inhibited.  Since vaccines activate the B cells to secrete antibody, the cytotoxic (killer) T cells are subsequently suppressed.  (In fact, progressive vaccinia (following vaccination with smallpox) occurs in the presence of high titers of circulating antibody to the virus1 combined with suppressed cytotoxic T cells, leading to spreading of lesions all over the body).  This suppression of the cell mediated response is thus a key factor in the development of cancer and life threatening infections.  In fact, the “prevention” of a disease via vaccination is, in reality, an inability to expel organisms due to the suppression of the cell-mediated response.  Thus, rather than preventing disease, the disease is actually prevented from ever being resolved.  The organisms continue circulating through the body, adapting to the hostile environment by transforming into other organisms depending on acidity, toxicity and other changes to the internal terrain of the body as demonstrated by the works of Professor Antoine Béchamp.  He established this prior to the development of the “germ theory” of disease by Louis Pasteur.  Pasteur’s “germ theory” was a plagiarist’s attempt to reshape the truth from Béchamp into his own “original” premise – the beLIEf that germs are out to “attack” us, thereby causing dis-ease. Thus, treatment of infection with antibiotics as well as “prevention” of disease with vaccines are both just corrupted attempts at cutting off the branches of dis-ease, when the root of the cause is a toxic internal environment combined with nutritional deficiency.  However, since Pasteur’s germ theory was conducive to the profits of the burgeoning pharmaceutical cartels that only manage dis-ease, no mention of the work of Professor Béchamp is made in medical school curricula.

     To make matters worse than the suppression of cellular immunity which occurs when vaccines are injected, adjuvants (which are substances added to vaccines to enhance the antibody response) can actually lead to serious side effects themselves. Adjuvants include oil emulsions, mineral compounds (which may contain the toxic metal aluminum), bacterial products, liposomes (which allow delayed release of substances), and squalene.  The side effects of adjuvants themselves include hyperactivity of B cells leading to pathologic2 levels of antibody production,  as well as allergic reaction to the adjuvants themselves (as demonstrated in Gulf War I soldiers injected with vaccines containing the adjuvant squalene, to which antibodies were found in many soldiers). Note that the pathologically elevated hyperactivity of antibody production caused by adjuvants also results in a distraction from the other antigens that the immune system encounters “naturally”, which must be addressed to maintain health.




When a B lymphocyte encounters an antigen, it is stimulated to mature into a plasma cell, which then produces antibodies (also called immunoglobulins, or Ig). Antibodies protect the body by helping other immune cells ingest antigens, by inactivating toxic substances produced by bacteria, and by attacking bacteria and viruses directly. Antibodies also activate the complement system. Antibodies are essential for fighting off certain types of bacterial infections.

Each antibody molecule has two parts. One part varies; it is specialized to attach to a specific antigen. The other part is one of five structures, which determines the antibody’s class-IgG, IgM, IgD, IgE, or IgA. This part is the same within each class.

IgM: This class of antibody is produced when a particular antigen is encountered for the first time. The response triggered by the first encounter with an antigen is called the primary antibody response. Normally, IgM is present in the bloodstream but not in the tissues.

IgG: The most prevalent class of antibody, IgG is produced when a particular antigen is encountered again. This response is called the secondary antibody response. It is faster and results in more antibodies than the primary antibody response. IgG is present in the bloodstream and tissues. It is the only class of antibody that crosses the placenta from mother to fetus. The mother’s IgG protects the fetus and infant until the infant’s immune system can produce its own antibodies.

IgA: These antibodies help defend against the invasion of microorganisms through body surfaces lined with a mucous membrane, including those of the nose, eyes, lungs, and digestive tract. IgA is present in the bloodstream, in secretions produced by mucous membranes, and in breast milk.

IgE: These antibodies trigger immediate allergic reactions (see Allergic Reactions: Introduction). IgE binds to basophils (a type of white blood cell) in the bloodstream and mast cells in tissues. When basophils or mast cells with IgE bound to them encounter allergens (antigens that cause allergic reactions), they release substances that cause inflammation and damage surrounding tissues. Thus, IgE is the only class of antibody that often seems to do more harm than good. However, IgE may help defend against certain parasitic infections that are common in some developing countries.

IgD: Small amounts of these antibodies are present in the bloodstream. The function of IgD is not well understood.

An Introduction to Immunity


INNATE IMMUNITY = This can best be described as GENETIC IMMUNITY or that immunity an organism is BORN WITH. This type of immunity can be an immunity that applies to the vast majority of the members of a species (SPECIES IMMUNITY), or it can be an immunity that applies to only a certain subgroup within a species down to a few individuals within that species. For example, cattle suffer from the cowpox virus, but appear to have a SPECIES IMMUNITY to the closely related smallpox viruses, whereas smallpox is a deadly disease to humans , but cowpox is a mild localized skin infection. Humans are susceptible to the HIV virus, but most of our related primates are immune to HIV, but they suffer from HIV-like viruses to which we appear to be immune. Within a species there may exist SUBGROUPS that are STATISTICALLY immune or resistant to particular pathogens. For example, the Northern Europeans appears to be more resistant to tuberculosis than are most Africans, whereas Africans are naturally resistant to a variety of African diseases that readily kill the “whites”. Finally, because of the genetic variation within every species INDIVIDUALS are statistically more resistant to some diseases, and more susceptible to other diseases. Most of you know those within your own families that “rarely” get colds or the flu, while other family members catch one respiratory infection after another. While there are many factors (diet, stress etc.) that could explain these individual differences, one of them is that certain COMBINATIONS OF GENES render some more resistant to the common cold viruses, whereas others of us are very susceptible. This type of immunity has NOTHING TO DO WITH the type of specific immunity we are discussing in this section.

ACQUIRED IMMUNITY = This refers to immunity that one acquires in one of two ways, active or passive. These are subdivided into the following further categories:

a) ACTIVE NATURALLY ACQUIRED IMMUNITY = This occurs when individuals suffer from
    a natural infection of a pathogen and become immune to that pathogen upon recovery (e.g.
b) ACTIVE ARTIFICIALLY ACQUIRED IMMUNITY = This occurs when individuals are
    actively vaccinated with an antigen that confers immunity.

c) PASSIVE NATURALLY ACQUIRED IMMUNITY = This occurs when individuals receive
    antibodies from their mother by a natural process, such as in BREAST MILK or in-utero transfer of
    antibodies from mother to fetus. In mammals, mother’s milk is know to contain a large concentration
    of antibodies and other antiviral and antibacterial substance that protect the newborn infants. Further,
    the mother’s antibodies cross the placental barrier, particularly near the end of term. In both these
    circumstances the infant is only resistant to whatever the mother is resistant to.
d) PASSIVE ARTIFICIALLY ACQUIRED IMMUNITY = This occurs when individuals are
    injected with POOLED serum from immune individuals that contain antibodies against a large number
    of pathogens. In the case of humans, a fraction of blood serum, GAMMA GLOBULIN, that is
    highly enriched in antibodies is injected into individuals that have been exposed to certain pathogens.
    The GAMMA GLOBULIN is obtained from pooled sera from many individuals and thus contains a
    broad spectrum of antibodies.



PASSIVE acquired immunity is short lived as the antibodies eventually die off or are themselves removed from the body as foreign protein. Since the person receiving the passive dose DOES NOT PRODUCE their own antibodies, the immunity is TRANSIENT.

The ACTIVE forms of immunity are generally long lived, particularly in the case of recovery from a CLINICAL INFECTION. Sometimes this immunity it lifelong, but in other cases it is not. Vaccinations may induce long-lived immunity, but recent data indicate that vaccinations may not last as long as once was hoped. For example, there is a very effective vaccine against tetanus, but it lasts only a few years and every year hundreds of people who have been vaccinated against this bacterium die because they have not gotten their BOOSTER SHOTS (vaccinations given periodically to booster the immunity of previous vaccinations) every three to five years.


Vaccines and the immune response to vaccination

  • Live and live-attenuated vaccines Live vaccines contain either low doses or doses of mild forms of the disease organism. Live-attenuated vaccines contain living disease organisms that have been treated in some way to reduce their ability to cause disease while still causing an immune response. Both of these vaccines contain living organisms that are able to infect and multiply in the host and this enhances the strength and duration of the immune response.


  • Killed (or inactivated) vaccines Killed contain high doses of the killed disease organism. Killed vaccines generally result in a weaker and shorter immune response than live vaccines due to their inability to infect and multiply in the host.


  • Sub-unit vaccines These vaccines contain doses of purified antigens extracted from the disease organism.


  • Recombinant vaccines These vaccines are produced by incorporating the DNA for the antigens that stimulate a disease response to a disease organism into a vector (or carrier), such as a harmless virus, which is then used as a live vaccine.


  • DNA vaccines These vaccines contain purified DNA for the antigens that stimulate an immune response to a disease organism.


  • Conjugate vaccines These vaccines are used to elicit an immune response to an antigen that is normally able to evade detection by the immune system. They contain the antigen bound to a compound, such as a protein, to form a complex that is detectable by the immune system.


Antibody titers and immunity: Are they related?

Dr. Tedd Koren, D.C. stated, “Whenever we read vaccine papers, the MD researchers always assume that if there are high antibody levels after vaccination, then there is immunity (immunogencity). But are antibody levels and immunity the same? No! Antibody levels are not the same as IMMUNITY. The recent MUMPS vaccine fiasco in Switzerland has re-emphasized this point. Three mumps vaccines-Rubini, Jeryl-Lynn and Urabe (the one withdrawn because it caused encephalitis)- all produced excellent antibody levels but those vaccinated with the Rubini strain had the same attack rate as those not vaccinated at all, there were some who said that it actually caused outbreaks.” [Ref: Schegal M et al Comparative efficacy of three mumps vaccines during disease outbreak in Switzerland: cohort study. BMJ, 1999; 319:352-3.]

According to Trevor Gunn, B.Sc., “Many measles vaccine efficacy studies relate to their ability to stimulate an antibody response, (sero-conversion or sero-response). An antibody response does not necessarily equate to immunity….the level of antibody needed for effective immunity is different in each individual….immunity can be demonstrated in individuals with a low or no detectable levels of antibody. Similarly in other individuals with higher levels of antibody there may be no immunity. We therefore need to stay clear on the issue: How do we know if the vaccine is effective for a particular individual when we do not know what level of antibody production equals immunity?”

Dr. John March, a developer of animal vaccines, wrote, “Particularly for viral diseases, the ‘cellular’immune response is all important, and antibody levels and protection are totally unconnected.”

It is clear that immunity does not come from antibodies or even ‘memory cells’, although memory cells may play a small part in the much larger processes of protecting health. If a person is healthy, first time natural exposure to a virus does not necessarily result in disease. In fact, the majority of first time exposures result in no symptoms but do result in ‘antibodies’ which ‘prove the exposure’ but also prove that immunity was present before the exposure. Total body health is the only true immunity. The concept that immunity comes from ‘memory cells’ is none-the-less valuable in that it points out that booster shots are totally unnecessary. Knowing that total health equals immunity is a basic key to understanding that vaccinations are unnecessary and ineffective.


Titers: What do they tell us?

A “titer” is a measurement of how much antibody to a certain virus (or other antigen) is circulating in the blood at that moment. Titers are usually expressed in a ratio, which is how many times they could dilute the blood until they couldn’t find antibodies anymore. So let’s say they could dilute it two times only and then they didn’t find anymore, that would be a titer of 1:2. If they could dilute it a thousand times before they couldn’t find any antibody, then that would be a titer of 1:1000.

A titer test does not and cannot measure immunity, because immunity to specific viruses is reliant not on antibodies, but on memory cells, which we have no way to measure. Memory cells are what prompt the immune system to create antibodies and dispatch them to an infection caused by the virus it “remembers.” Memory cells don’t need “reminders” in the form of re-vaccination to keep producing antibodies.

Vaccine Titer Table

This just doesn’t apply to humans but pets as well.

Breast Milk and Antibodies

Antibodies, or immunoglobulins, are found in breast milk. There are five basic forms: IgG, IgA, IgM, IgD and IgE. All 5 forms have been found in human breast milk, but the most prevalent type is IgA, also known as secretory IgA. It is also found in large amounts throughout the intestinal tract and respiratory system of adults. Two joined

IgA antibodies protect the antibody molecules from being reduced by gastric acid and digestive enzymes present in the intestinal tract and stomach.


It takes several weeks or even months after birth for infants to make secretory IgA on their own. Through breast milk, secretory IgA molecules are passed on to the nursing baby and help in many ways beyond their natural ability to bind to microorganisms and keep them away from the body’s tissues. Bottle-fed infants do not have the advantage of fighting ingested pathogens until they can produce secretory IgA on their own.


The medical establishment know that infants who are breastfed contract fewer infections than formula-fed babies. Breast milk protects against E. coli, salmonellae, shigellae, streptococci, staphylococci, pneumococci, poliovirus, and rotaviruses.  It is known that infants who receive formula can contract more sickness, meningitis, infections of the intestinal tract, ear, respiratory tract and urinary tract than do breastfed babies.

Antibodies transmitted to an infant are targeted against germs in the baby’s surroundings. A mother will begin producing antibodies when she comes in contact with a disease-causing agent. Antibodies made by the Mother are specific to her environment. The baby will then receive protection from infectious germs it will encounter the most in the first few weeks of life. Antibodies passed to the baby will disregard the useful bacteria found in the gut. This gut flora is used to get rid of the growth of harmful organisms, which will provide another measure of resistance. Secretory IgA molecules, unlike other antibodies, ward off diseases without causing inflammation.

Several other molecules in human milk prevent microbes from attaching to mucosal surfaces. Oligosaccharides, which are simple chains of sugars, can intercept bacteria, forming harmless complexes that the baby excretes. Breast milk also contains mucins that contain protein and carbohydrate. They are also capable of attaching to bacteria and viruses and eliminating them from the body.

There are other helpful molecules present in breast milk. A  molecule of a protein called lactoferrin, can bind to two atoms of iron. Since many pathogenic bacteria thrive on iron, lactoferrin can stop their spread. It is especially effective at reducing or slowing down the proliferation of organisms that can cause serious illness in infants such as Staphylococcus aureus. One of the oldest disease-resistance factors known in breast milk is the Bifidus factor, which promotes the growth of a beneficial organism called Lactobacillus bifidus. Interferon, which is found in colostrum that a mother produces during the first few days after birth, can be thought of as an antiviral agent. Fibronectin which is present in colostrum, can minimize inflammation and aid in repairing tissue damage. Colostrum is a natural and 100% safe vaccine since it contains large quantities of secretory immunoglobulin A, or IgA.

Immune cells are also in abundance in breast milk. They consist of white blood cells, which fight infection and activate other defenses. Cells such as neutrophils act as phagocytes in the infant’s intestinal tract for about 2 months after birth. Macrophages are present in about 40 percent of all the leukocytes in colostrum. In some experiments they have shown they are better capable than are their counterparts in blood. Macrophages in breastmilk also manufacture lysozyme, which increases the amount in the infant’s gastrointestinal tract. An enzyme called Lysozyme destroys bacteria by disrupting their cell walls. Macrophages in the digestive tract can get lymphocytes to action against invaders. B lymphocytes raise antibodies and T lymphocytes kill infected cells directly or provide direction to other chemical messages that will mobilize other components of the immune system. Breast milk lymphocytes proliferate in the presence of Escherichia coli, which is a bacterium that can cause severe illness in babies. However, they are less responsive than blood lymphocytes to germs. Breast milk lymphocytes also produce gamma interferon, migration inhibition factor, and monocyte chemotactic factor. All of which can strengthen the immune response.

There are studies showing that breast milk may induce an infant’s immune system to mature more quickly. Breastfed babies produce higher levels of antibodies in response to immunizations. Certain hormones in milk like cortisol and proteins such as epidermal growth factor, nerve growth factor, an insulin-like growth factor, and somatomedin C, can close up the leaky mucosal lining of the newborn. This makes it impossible for harmful pathogens to get though. Other compounds in breast milk stimulate a baby’s own production of secretory IgA, lactoferrin and lysozyme but are not known. Secretory IgA, lactoferrin and lysozyme are all found in the urine of breastfed babies. Breastfed babies cannot absorb these molecules from breast milk into their intestinal tract. The molecules are produced in the mucosa of the baby’s urinary tract. Therefore, breastfed babies have a lower risk of acquiring urinary tract infections.

Breast milk has the ability to protect infants against infection until they can protect themselves, along with providing them with all the nutritional requirements they need. Immune protection continues to improve and change dependent upon the needs of the infant and age throughout the duration of breastfeeding no matter how long that may be.  A baby may not necessarily receive enough of their mother’s  IgG immunities through breast milk to qualify as an immunization against a particular disease, but IgA, certain fatty acids, etc, in the breast milk active and do protect against illnesses. 
Dr. Jack Newman in How Breast Milk Protects Newborns, states: “Free fatty acids present in milk can damage the membranes of enveloped viruses, such as the chicken pox virus, which are packets of genetic material encased in protein shells.” The secretory IgA in breast milk also activates against the chicken pox virus in vitro.
As the baby grows, some of the immune factors in breast milk increase in concentration so older babies still receive plenty of immune factors. As a baby starts to nurse less and milk supply decreases, the concentration of immunities increases. [source: Goldman AS et al. “Immunologic components in human milk during weaning.” Acta Paediatr Scand. 1983 Jan;72(1):133-4.] 

List of Immune Factors In breast milk: 

alpha-Lactalbumin (variant) 
alpha2-macroglobulin (like) 
Bifidobacterium bifidum 
CCL28 (CC-chemokine) 
Chondroitin sulphate (-like) 
Complement C1-C9 
Free secretory component 
Fucosylated oligosaccharides 
Gangliosides GM1-3, GD1a, GT1b, GQ1b 
Glycolipid Gb3, Gb 
Glycoproteins (mannosylated) 
Glycoproteins (receptor-like) 
Glycoproteins (sialic acid-containing or terminal galactose) 
Haemagglutinin inhibitors 
Lactadherin (mucin-associated glycoprotein) 
Lewis antigens 
Milk cells (macrophages, neutrophils, B & T lymphocytes) 
Mucin (muc-1; milk fat globulin membrane) 
Nonimmunoglobulin macromolecules (milk fat, proteins) 
(Tri to penta) phosphorylated beta-casein 
Prostaglandins E1, E2, F2 alpha 
RANTES (CC-chemokine) 
Secretory IgA 
Secretory leukocyte protease inhibitor (antileukocyte protease; SLPI) 
Sialic acid-glycoproteins 
sialylated oligosaccharides 
Sialyloligosaccharides on sIgA(Fc) 
Soluble bacterial pattern recognition receptor CD14 
Soluble intracellular adhesion molecule 1 (ICAM-1) 
Soluble vascular cell adhesion molecule 1 (VCAM-1) 
Sulphatide (sulphogalactosylceramide) 
Trypsin inhibitor 
Vitamin A 
vitamin B12 
Xanthine oxidase (with added hypoxanthine) 
Unidentified factors