What are your chances of Polio?

Susceptibility of Polio was determined by several things. Sick kids never got polio, as weird as that sounds. People who are the ‘life of the party’, over-worked, or who run on never-ending batteries, were dead sitting ducks for polio, but was also dependent on their diet. Healthy people, who if incubating Polio got stressed, pregnant, were overworked, or got vaccinated, could come down with Polio. Immunodeficiency could potentially make polio more fatal. Tonsillectomies guaranteed a higher chance of getting polio. In 1994 it was finally known that Selenium status made a difference.  Cuba got struck with what was first thought to be polio, but which was eventually found to be a coxsackie B virus. Certain sectors of Cuba did not get any paralysis at all, though they were found to have the same infection quota of the virus. Studies found that the mineral and vitamin content of the food people ate had protected them against the paralysis factor of the virus. There are some viruses, like coxsackie, that give guinea-pigs an identical syndrome to polio.

In guinea pigs it is known that there is an epigenetic factor, which means, that if a guinea pig with that gene susceptibility is exposed to that virus in certain nutritional deficiency situations they will get that disease. Polio does not need a human host. See Pubmed.


A serological indication of the existence of a guineapig poliovirus.


Attempts were made to clarify whether laboratory guineapigs may harbour a poliovirus which, in 1911, was described as the cause of a disease called guineapig lameness. By the use of ELISA for antibodies against the poliovirus, Theiler’s murine encephalomyelitis virus (TMEV), it was shown that two pet shop guineapigs suffering from lameness had extremely high titres against poliovirus, while healthy guineapigs from the same pet shop were negative. Clearly positive results were also found in 35 out of 152 laboratory guineapig sera. Positive results were found in only two out of six breeding centres, but in three out of three experimental units, all of which purchased guineapigs from one of the seropositive breeding colonies. The diseased guineapigs recovered fully after treatment with vitamins in the drinking water, a treatment used for guineapig lameness by small animal practitioners. A theory that vitamin C deficient guineapigs are, due to an impaired steroid secretion, predisposed to succumbing to infection and develop demyelinating disease similar to that in TMEV infected mice is discussed briefly. Guineapig sera were also tested serologically for other infections. Antibodies against lymphocytic choriomeningitis virus, Clostridium piliforme and Toxoplasma gondii were not found, but one breeding colony was infected with adenovirus, pneumonia virus of mice, reovirus type 3, Sendai virus, parainfluenza (simian) virus type 5 and Encephalitozoon cuniculi. Two other breeding colonies were infected with both reovirus type 3 and E. cuniculi. In all three experimental units infection with adenovirus was observed, and in two of these Sendai virus and E. cuniculi antibodies were also found. The pet shop guineapigs were infected with adenovirus, reovirus type 3 and E. cuniculi.


Nutrition was the primary prevention and doctors used nutrition programs to get people over polio. All is dependent on how malnourished your body is, amount of toxins and pesticides, and other your system is full of. The higher the Vitamin C deficiency, the more devastating the disease can be.


Polio and Herd Immunity

The polio vaccine in U.S. was not attributed to herd immunity. Research done prior to the use of the vaccine stated that in any epidemic 98.2% of people already had antibodies. Only 36% of people in the USA had had the primary 3 shots of SALK by 1958. The majority of these people did not need the vaccine, and the small herd effect of 36% of the population that was vaccinated is also negligible. The AMA removed all copies of the Polio Surveillance Units Reports which clearly showed that up until 1962, the percentage of paralytic and non-paralytic polio was more in the vaccinated community than the unvaccinated. 


Let’s go back through time and take a look at some issues …


If you look at the oldest graphs, they show that most polio disappeared by the time less than half the U.S. had been vaccinated the first time, yet everyone had to be revaccinated?  

How did the definition of paralytic polio differ in 1953 from the one in 1955, 1956 and 1959? Why were those changes made? 

When vaccinated people got polio within 30 days of a vaccine it was not considered to be related to the vaccine, when the allowable incubation time is relevant? 
All Salk vaccine were Live, not inactivated. The primary use of OPV during epidemics isn’t to create herd immunity. It is used to disrupt the circulation of all enteric viruses by filling a gap and to try to prevent the spread.


 Symptoms of Polio


Headache and general malaise; gastro-intestinal disturbance (i.e. diarrhea, vomiting); sore throat; stiffness of neck and back; aching muscles. On the fourth or fifth day, if muscles are affected, paralysis may spread during the following 36 hours. Coughing, swallowing or speech may be affected. As with any disease, susceptibility is the key.


Treatment and Prevention

The number one preventative measure is to avoid contaminated food and water. The second most important preventative measure is to ensure a healthy digestive system. Rest in bed; physiotherapy can help muscles recover; homeopathic remedies can treat many of the symptoms associated with Polio. Gelsemium is the main homeopathic remedy, for its action on muscles and motor nerves. Recovery occurs in most cases.




Hiding Polio:


Viral or aseptic meningitis, Guillaine-Barré Syndrome (GBS), Chinese paralytic syndrome, chronic fatigue syndrome, epidemic cholera, cholera morbus, spinal meningitis, spinal apoplexy, inhibitory palsy, intermittent fever, famine fever, worm fever, bilious remittent fever, ergotism, encephalomyeloradiculoneuritis, post-polio syndrome, acute flaccid paralysis.




CFS, also known as, Myalgic Encephalomyelitis. Myalgic means Muscle and encephalo means brain. myelitis means inflammation of the covering of the nerves.

polio is contracted from ingesting the virus, which then goes to the small intestine and reproduces there. With VACCINES, particularly the OPV Sabin vaccine, the traditional polio viruses were replaced by other members of the same family called Coxsack e viruses.

When the Coxsackie viruses were isolated from cfs patients, they didn’t realize that it was a new form of polio. This new polio was caused by the replacement of the polio viruses with their partners, the Coxsackie viruses. the researchers didn’t get the connection at first. the new polio cases were labled post-polio syndrome, chronic fatigue syndrome, or myalgic encephalomyelitis.

Modern genetics has confirmed the genetic similarity between polio viruses, Coxsackie, and a group called the Echo viruses. Before the invention of the Salk and Sabin vaccines, there were only three polio viruses. Now, with the alterations of the human gut over the years as a result of these vaccines, there are at least 72 viral strains that can cause polio-like diseases.

the evidence of Polio Changing rather than the elimination of IT is not new.  After the introduction of the vaccines, the trends of NEW polio INCREASED and it has been recognized by neurologists for 40 years. The terms atypical and abortive polio have been kept quiet because it would point to the fact that polio is more common than lead to believe and caused by polio vaccination.

infantile paralysis as polio was called then, has become in the modern era adult paresis (muscle weakness).  Dr. Elizabeth Dowsett, a microbiologist from Britain. states: True CFS “strikes one clinically as being polio-like and it has often been diagnosed as ‘nonparalytic polio.” Dowsett says the term chronic fatigue syndrome was “an unfortunate mistake” because it is a neurological disease and the doctors waiting six months before doing anything so they could label it “chronic,” reuined the chances of identifying the virus. the patient now has chronic new-age polio that will not be amenable to treatment.


If CFS is a form of polio, and 72 viruses are in everyones intestine, then why doesn’t everyone come down with CFS? NOT EVERYONE will come down with a disease simply because they are exposed to it. Some people have stronger immune systems than others and that is where good nutrition and hygiene come in. During the polio epidemic of the ’30s and ’40s, most of the children who had polio, didn’t even know they had it. It was passed off as a cold. Multiple sclerosis, amyotrophic lateral sclerosis, CFS, Tourette syndrome, learning disabilities, Guillain Barre Syndrome, idiopathic epilepsy, and other neurological conditions may very well be forms of polio induced by these vaccines. The Salk and Sabin vaccines opened a Pandora’s box as we now have 72 types of polio rather than three!




In the U.S. DDT was used during polio epidemics. It was thought that polio was spread by flies and mosquitoes. In areas where DDT was used, polio increased instead of decreased. The U.S. was spraying it everywhere, so much so, even cows got polio. The description of polio matched symptoms of DDT poisoning as well. In Sweden and Sicily the same thing happened. Then there is epigenetics. Dr. Wyatt wrote two medical articles which talked about genetic susceptibility to polio.