Tetanus vaccine is used as a ‘just in case’ scenario if the spores start replicating and producing a deadly toxin. This toxin can get into the bloodstream and then the central nervous system. The spores do not cause the disease; the toxin does. Tetanus can survive anywhere as a spore. The spore is incapable of causing illness in the presence of oxygen. Any wound that is getting a good supply of oxygen won’t allow the bacteria to thrive and begin to secret toxin. A minor wound like a scrape will be oxygenated just because it’s open to the air. A puncture wound should receive oxygen through circulating blood, so even puncture wounds shouldn’t be a problem for healthy children and adults with good circulation, as long as you clean them well. Burns and crushing wounds are more likely to become infected with tetanus, especially burns, since those types of injuries can destroy blood vessels in the area.
When a person gets tetanus from an injury (deep puncture wound that doesn’t bleed) and recovers, he is not immune. We can get tetanus repeatedly in our lives. If the body can not build immunity from a natural toxin, how can we expect the vaccine to do that?
A 1969 article from the New England Medical Journal, Volume 280, Number 11, March 13, page 570, is an interesting decline graph for mortality rates. It shows that the mortality (death) rate plummeted dramatically from 64/100,000 in 1900 to 8/100,000, in 1940. By 1950, with most mothers still unvaccinated, it was 4.5/100,000. They say that it may have been the use of anti-toxin from 1923. Well, the antitoxin can kill all by itself, because it is made in horses, and has horrendous side-effects of its own. Some people treated with anti-tetanus toxoid, will die of the toxoid side-effects. There are plenty of people around in the USA who have never been vaccinated, including the Amish, who work with horses (horse manure is conductive to tetanus) and they haven’t died out as a community.
An interesting study in the American Journal of Public Health, August 1984, Vol 74, No 8, reported that in 1,900 adults over 20 years of age, the overall percentage immunized was 38.6%. So where are all the unimmunized adults dying from tetanus again?
According to Center for Disease Control (CDC) tetanus vaccine experts, The 1988 to 1991 serosurvey indicated that 20 per cent of children 10 to 16 years of age did not have a protective level of antibody. A 1979 study found that in a sample of 1900 adults over 20 years of age, only 386 per cent were fully immunised. If we extrapolate from that study alone, about 120 million or so citizens (60 per cent of 200 million) were unprotected yet virtually none of them was getting tetanus, let alone dying from it. Walene James, in her book Immunization: the Reality Behind the Myth, points out that in the United States in 1990 there were 25,700 cases of tuberculosis with 1800 deaths, tuberculosis therefore immensely outweighing tetanus as a cause of death. (Mothers of unvaccinated children who might be worried about them contracting tetanus because theyve just joined the pony club, take note!) In the United States, with an average of seven to 10 deaths per year from tetanus, there is a 180 to 260-times greater chance of dying from tuberculosis. In fact, since lightning strikes about 1800 people a year in that country, with an approximate mortality rate of 25 per cent (450 deaths), there is a 45-times greater chance of being killed by lightning than tetanus! (Ref: Tetanus Vaccination by Jason Sanders Issue, 97 Page, 22)
The tetanus vaccine takes 1-3 weeks to take effect, which is longer than the incubation period for tetanus. The first 1-2 doses of tetanus vaccine does not confer any immunity, but simply “primes the immune system” to respond to future doses, according to the CDC. If you are concerned about a specific injury/wound, the immunoglobulin confers immediate immunity for the duration of the incubation period. The immune globulin is blood/fluid which already contains antibodies to tetanus. The immunoglobin is also a risk since it’s a blood product so you would want to sure it was warranted.
“Tetanus immune globulin is not a vaccine. It is a preparation that is made from serum (part of the blood) from a person or animal (such as a horse) that contains antibodies against tetanus. It provides immediate, short-term protection against the disorder, but does not provide long-term immunization. It can be used when someone is believed to have been exposed to the bacteria…” …tetanus toxoid is of no value unless the individual has previously been vaccinated, since a primary antibody response takes at least 14 days, and the incubation period of the disease can be considerably shorter than this (three to 14 days).
“..tetanus toxoid is of no value unless the individual has previously been vaccinated, since a primary antibody response takes at least 14 days, and the incubation period of the disease can be considerably shorter than this (three to 14 days)..If you do decide to vaccinate your children with tetanus toxoid alone, there is no need to vaccinate until the child is old enough to walk around and navigate on his or her own (18 to 24 months), at which time the vaccine is far less likely to cause complications.”
“The incubation period (for tetanus) varies from 3 to 21 days, usually about 8
days. In general the further the injury site is from the central nervous system, the longer the incubation period. The shorter the incubation period, the higher the chance of death.”
It’s not just antibodies because any blood product has to be treated because since blood contains a lot of things other than just red cells. Viruses and bacteria can get in there as its part of the immune system which is why they take the antibodies from blood.
A good read:
Normal serum contains IgG, IgM, and IgA antibodies, which are referred to as natural antibodies because they are induced without deliberate immunization and are independent of antigenic exposure. They are considered key to the immunoregulatory effects of immune globulin in immune-mediated disorders (Kazatchkine, 2001). Natural autoantibodies appear to be more polyreactive than immune antibodies; natural antibodies can frequently bind to different antigens (Kazatchkine, 2001). Natural autoantibodies can (1) bind to pathogens; (2) help remove senescent or altered molecules, cells, and tumors; (3) induce remyelination; and (4) inhibit the growth of autoreactive B-cell clones.IVIG contains cytokines, antibodies of unclear clinical significance, perhaps neutralizing; interestingly, antibodies against granulocyte macrophage colony-stimulating factor, interferon, interleukin 1, and interleukin 6 in immune globulin have biologic activity in vivo (Kazatchkine, 2001). IVIG contains natural antibodies, accounting for some of its effects.
The IVIG that is available contains complete IgG molecules. The IgG subclasses match those in normal human serum. Most preparations contain trace amounts of IgA, which can sensitize IgA-deficient persons during long-term treatment. Immune globulin also contains trace amounts of cytokines, soluble CD4, CD8, and HLA molecules.
Prevalence of hepatitis C virus in plasma pools and the effectiveness of cold ethanol fractionation.
Screening blood donations for antibodies against hepatitis C virus (HCV) greatly reduces the risk of transmitting HCV by transfusions. However, despite such screening programs, plasma pools still contain a high percentage of HCV ribonucleic acid as determined by polymerase chain reaction. This result would not be alarming if the procedures for producing blood products included steps to inactivate or remove HCV. Although this appeared to be the case for all blood products, such as coagulation factors and most immunoglobulins, which are subjected to an inactivation step, the effectiveness of the cold ethanol fractionation process still needed to be determined. In validation experiments using bovine viral diarrhea virus as a model virus for HCV, we demonstrated that the Cohn-Oncley cold ethanol fractionation process neither inactivated nor removed this virus sufficiently. Our observations may help to explain how HCV was transmitted to a number of recipients of intravenous immunoglobulin.
Tetanus Vaccination by Dr Mendelsohn MD (The People’s Doctor Newsletter 1976-1988)
1) Scientific evidence shows that too—frequent tetanus boosters actually may interfere with the immune reaction.
2) There has been a gradual retreat of even the most conservative authorities from giving tetanus boosters every one year to every two years to every five years to every 10 years (as now recommended by the American Academy of Pediatrics), and according to some, every 20 years. All these numbers are based on guesses rather than on hard scientific evidence.
3) There has been a growing recognition that no controlled scientific study (in which half the patients were given the vaccine and the other half were given injections of sterile water) has ever been carried out to prove the safety and effectiveness of the tetanus vaccine. Evidence for the vaccine comes from epidemiologic studies which are by nature controversial and which do not satisfy the criteria for scientific proof.
4) The tetanus vaccine over the decades has been progressively weakened in order to reduce the considerable reaction (fever and swelling) it used to cause. Accompanying this reduction in reactivity has been a concomitant reduction in antigenicity (the ability to confer protection). Therefore, there is a good chance that today’s tetanus vaccine is about as effective as tap water.
5) Until the last few years, government statistics admitted that 40 percent of the child population of the U.S. was not immunized. For all those decades, where were the tetanus cases from all those rusty nails?
6) There now exists a growing theoretical concern which links immunizations to the huge increase in recent decades of auto—immune diseases, e.g., rheumatoid arthritis, multiple sclerosis, lupus erythematosus, lymphoma, and leukemia. In one case, Guillain—Barre paralysis from swine flu vaccine, the relationship turned out to be more than just theoretical.
In preparing my courtroom testimony on behalf of a child who allegedly was brain—damaged as a result of the DPT (diphtheria, pertussis, tetanus) vaccine, I reviewed the prescribing information (package insert) for the Connaught Laboratories product which was administered to this child. The 1975 and 1977 package insert information which measured seven—and—a—half inches long listed three scientific references in support of the indications, contraindications, warnings, cautions, and adverse reactions to this vaccine. By 1978, the length of the insert had grown to 13 1/2 inches, and the number of scientific references had increased to 11. By 1980, the package insert was 18 inches long, and the references numbered 14. Of those newly—added references, seven (three from U.S. medical journals and four from foreign medical journals) dealt specifically with reactions to the tetanus DPT portion of the (toxoid) vaccine.
The Journal of Allergy and Clinical Immunology, 1973, carried an article entitled “Hypersensitivity to Tetanus Toxoid,” and in a volume entitled “Proceedings of the II International Conference on Tetanus” (published by Hans Huber, Bern, Switzerland, 1967), an article appeared entitled “Clinical Reactions to Tetanus Toxoid.”
Basics:
1. A wound that bleeds can not grow tetanus
2. Tetanus vaccine creates no reaction in the body for 3 weeks while tetanus grows within 10 days (but never in a wound that bleeds!)
3. The body can not build immunity to the poisons nor the vaccine
4. Children do not get tetanus. Their circulatory system is designed that way. (Only exception is in Africa where the umbilical stump is covered with mud.)
Health Measures-
-Use hydrogen peroxide. It cleans out the wound and the bubbles provide the oxygen needed.
– Let the wound bleed.
Overuse of the vaccine can result in hypersensitivity to tetanus and more severe infection-as documented in those with recent and/or over-frequent booster doses.
Immunity to tetanus: tetanus antitoxin and anti-BIIb in human sera.
Severe tetanus in immunized patients with high anti-tetanus titers
Relation between protective potency and specificity of antibodies in sera of tetanus immunized individuals
Response to single dose of tetanus vaccine in subjects with naturally acquired tetanus antitoxin
Naturally acquired tetanus antitoxin in the serum of children and adults in Mali
Naturally acquired immunity to tetanus toxin in an isolated community
New concepts on tetanus immunization: naturally acquired immunity
This issue was raised by a Roman Catholic priest in Mexico. He stated that many of the vaccinated women were miscarrying or could no longer conceive. The vaccine was tested by three groups and found to have had hcg in it. Women in other countries had the same thing happen. WHO got involved and shoved it all under a rug and denied everything. But think about it…if the focus was on tetanus protection for all of the people, why were only woman getting vaccinated? The 1995 BBC Horizon program was called “The Human Laboratory”, and was never shown in USA.
The same BBC documentary reported that women in the Philippines and Mexico have also been used as guinea pigs for a new experimental pregnancy vaccine. The HCG vaccine makes a woman’s body reject new pregnancies. …, it has been administered, without the consent or knowledge of patients, as a “piggyback” vaccine in a series of tetanus vaccine programs.
…But when women who had recently received the “tetanus vaccine” began having an inordinate number of miscarriages, this bureaucratic curiosity turned into charges of conspiracy. Subsequent lab tests of the tetanus vaccine confirmed it had indeed been laced with an HCG vaccine.
Subacute tetanus:
The clinical recognition of subacute tetanus.
The clinical features of a modified form of tetanus, termed ‘subacute tetanus’, occurring in non-immune patients are presented as manifested in five patients. Subacute tetanus has a good prognosis and favourable outcome. Trismus and abdominal rigidity may be minimal or absent. Nocturnal, brief generalized muscle spasms, palpably contracted sternomastoid muscles and spastic limbs are common features of the disease. The pathophysiology of the nocturnal muscle spasms requires elucidation.
Now compare it with this: Tetanus of immunized children
Five children aged five to 15 years contracted tetanus in Finland between 1969 and 1985, together with 101 adults. Four of the five had been adequately immunized against tetanus. The clinical picture of tetanus was mild or moderate, and none of the children needed respirator treatment. Epilepsy, meningitis and psychogenic symptoms were considered in the differential diagnosis. The course of tetanus in immunized patients is atypical and often benign, but the diagnosis is problematic–in contrast to affected children in developing countries, whose populations are not adequately immunized and where neonatal tetanus is common and often fatal.
It is mainly elderly people who die from tetanus. There is evidence that suggests that poor nutrition and lifestyle habits that impedes the immune system, which is connected to susceptibility, and usually in tandem with the age factor. Improved wound care has also greatly improved from the early 19th century.
The small minority of people who develop tetanus from trivial wounds are and the statistics are suggestive likely to be immune deficient, either because of old age, chronic ill-health, poor diet or drug taking (I include smoking and heavy drinking in this category) and most likely a combination of these factors. Many elderly people, as a result of poor appetite, have a diet lacking in essential vitamins and minerals. The same goes for intravenous drug users, another group prone to tetanus.Linus Pauling, double Nobel Laureate scientist and expert on vitamin C, believed sub-clinical scurvy from vitamin C deficiency was widespread amongst senior citizens, making them prone to many illnesses. Vitamin C is a nutrient that is critical for immunity, so it should perhaps come as no surprise to learn it can be specifically curative. Doctor Fred Klenner, a North Carolina physician, outlined in various papers published from 1948 to 1974 his success with using intravenous mega-doses of ascorbate to deactivate tetanus spores and their toxin. This makes sense, because vitamin C removes toxins from the bloodstream while also enhancing white blood cell activity. In addition, since vitamin C is vital to collagen formation and has been proven to speed wound healing time, it is possible it also helps the body isolate and contain tetanus at the wound site. (Wounds can apparently break down if the body lacks vitamin C in the tissues.) Perhaps this is one reason why smokers may be a little more prone to tetanus, since they are known to have less vitamin C in the body. Vaccine researcher Hillary Butler says she has personally known only two people who contracted tetanus and both were middle-aged people who drank and smoked heavily and had poor diets.
Tetanus vaccination makes the body unsusceptible to the disease by inducing production of neutralising antibody (or antitoxin) to the tetanus toxin; this is the result of introducing weakened toxin into the body (the vaccine contains no attenuated bacilli). Therefore, it seems ludicrous to suppose sub-clinical (non-disease manifesting) contact with the bacilli such as we all must be experiencing regularly can’t do the same. Indeed, the comprehensive and authoritative Vaccines edited by S.A. Plotkin and W.A. Orenstein alludes to this: Studies in the developing world and some developed nations … have shown substantial proportions of some reportedly unimmunized populations … [to have] detectable levels of antitoxin. Specifically, up to 80 per cent of people in India and up to 95 per cent of people in a group of Ethiopian refugees had levels of antitoxin [considered protective]. However, these pro-vaccine establishment authors dismissed the findings by concluding, Even if natural immunity occurs in some unimmunized populations, it has no substantial importance in the control of tetanus.
In the case of tetanus, while better general health as a result of social change has to be a factor in the declining mortality rate, the most important reason was that wound care techniques and sterilization procedures greatly advanced in this period. It meant fewer women contracted tetanus after giving birth, less people contracted it from surgery and far fewer babies contracted it when the umbilical cord was cut. According to Plotkin and Orenstein, in the United States there were 90 per cent less tetanus deaths occurring in babies in 1930 than in 1900. Today, the World Heath Organization estimates that 400,000 babies in the Third World die each year of tetanus because of the use of unsterile cutting instruments and poor neonatal care while the umbilical stump heals. (Ref: Tetanus Vaccination by Jason Sanders Issue, 97)
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